Tuesday, 22 July 2014

The Biochemistry of Argininosuccinate Lyase Deficiency Part III: The Dangers of Ammonia

Argininosuccinate lyase deficiency is a deficiency of the urea cycle. A result of this metabolic disorder is hyperammonemia, or elevated ammonia levels.

Ammonia is a highly toxic biochemical by-product which is the result of the metabolism of certain amino acids. In a person with ASL Deficiency, the body is incapable of completing the process of converting ammonia into urea, a less toxic form which can be easily removed from the body primarily through urine.

Ammonia is harmful enough when it is encountered in the environment, capable of causing burns on exposed surfaces. On the inside of the body, it is capable of causing even more problems. Elevated ammonia levels in the blood acts as a toxin and may affect the brain, resulting in a number of symptoms, some of which include confusion, lethargy, and change in mood, personality and behaviour among others, all of which are symptoms of Argininosuccinate Lyase Deficiency.


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